Looking for online definition of Fiessinger-Leroy-Reiter syndrome in the Medical Dictionary? Fiessinger-Leroy-Reiter syndrome explanation free. What is. Download Citation on ResearchGate | Fiessinger Leroy Reiter syndrome | Whether it follows an attack of diarrhea or a venereal infection, the. Annales de Dermatologie et de Vénéréologie – Vol. – N° 5 – p. – Arthrite réactionnelle (syndrome de Fiessinger-Leroy-Reiter) – EM|consulte.

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List of medical eponyms with Nazi associations. Finger nails showing coarse pitting, yellowish discoloration, transverse ridges and subungual hyperkeratosis. D ICD – A number of physicians have suggested that the eponym is undeserved and use of the eponym has declined. In most patients, the symptoms last for months.

There was a history of improvement in the lesions and joint pain with ongoing treatment, but the lesions again flared up with self-stoppage of treatment for 6 months. The most common triggers are intestinal infections with SalmonellaShigella fiessknger Campylobacter and sexually transmitted infections with Chlamydia trachomatis. Differential diagnosis The differential diagnosis should include other forms of spondyloarthropathy ankylosing spondylitis, psoriatic arthritis, inflammatory bowel disease-associated spondyloarthropathy, juvenile-onset spondyloarthropathy see these termsand undifferentiated spondyloarthropathy.

Views Read Edit View history. Scandinavian Journal of Rheumatology. Keratoderma blenorrhagicum in a patient with Reiter syndrome. Most patients have severe symptoms lasting a few weeks to six months. Journal fiessniger Clinical Rheumatology.

Fiessinger-Leroy’s disease: A rare case report

Nail examination revealed coarse pitting, yellowish discoloration, transverse ridges, subungual hyperkeratosis, friability, and dystrophy [ Figure 5 ]. Bonnel aL. Madavamurthy P, Hanish VB. Manual of Rheumatology and Outpatient Orthopedic Disorders: Reactive arthritis ReA is an autoimmune disorder belonging to the group of seronegative spondyloarthropathies and is characterized by the classic triad of arthritis, urethritis and conjunctivitis. Occasionally, fiessingeg eruption is very widespread and it may evolve into generalized exfoliative dermatitis erythroderma.


Retrieved from ” https: Antistreptolysin-O titer and C-reactive protein were positive and rheumatoid factor was negative. The initial lesion is a dull, red macule which rapidly becomes papular and pseudovesicular. Detailed information Professionals Summary information Greekpdf Clinical practice guidelines Deutsch Access to the PDF text.

To conclude, Fiessinger-Leroy’s disease is a rare disease worldwide and hence we are reporting this case for documentation. Peron bF. The tendons and ligaments next to the joints may also be inflamed, particularly the Achilles tendon and the lower back joints.

Fiessinger-Leroy’s disease: A rare case report

You can move this window by clicking on the headline. Otherwise, treatment is symptomatic for each problem. Arora S, Arora G.

The mechanism by which the organisms causing infection in the urogenital or gastrointestinal tract lead to Fiessinger-Leroy’s disease in some individuals is not clear. Adult-onset Still’s lery Felty’s syndrome 3. It has been proposed that damaged exogenous pathogen-associated molecular patterns, derived from microbes, can disseminate through the pelvic and spinal lymphatic pathways and activate toll-like receptors TLRs.

Their activation triggers signaling pathways that result in the expression of immune response genes and cytokine production. Top of the page – Article Outline. A case of Reiter’s disease exacerbated by lithium. The arthritis often is coupled with other characteristic symptoms; this was previously referred to as Reiter’s syndrome, Reiter’s disease or Reiter’s arthritis.


Graves’ disease Myasthenia gravis Pernicious anemia.

Steroidssulfasalazine and immunosuppressants may be needed for patients with severe reactive symptoms that do not respond to any other treatment. Reiters syndrome — A case report and review of literature. Reiter allowed for experiments on concentration camp victims and the use of the term Reiter’s Syndrome has fallen out of favor. When reactive arthritis appears in a triad that also includes ophthalmic and urogenital manifestations, the eponym “Reiter’s syndrome” was often applied; German physician Hans Conrad Julius Reitera physician and leader of the Nazi party, described the condition in a soldier he treated during World War I.

However, recent human data suggest that TLR-2, not TLR-4, is important in determining reactive arthritis susceptibility after Salmonella infection. Outline Masquer le plan.

Arthrite réactionnelle (syndrome de Fiessinger-Leroy-Reiter) – EM|consulte

This is owing to the high frequency of the HLA-B27 gene in the white population. Heberden’s node Bouchard’s nodes. Furthermore, he was not the first physician to make associations between the arthritis and other symptoms: Age of onset varies widely, but there is a peak between 15 and 35 years of age. The main goal of treatment is to identify and eradicate the underlying infectious source with the appropriate antibiotics if still present.

Additional symptoms include scaly skin rashes on the hands or feet, nail changes, diarrhea, balanitis, fever, weight loss, and mouth ulcers. Ureaplasma and mycoplasma are rare causes.